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Kodak kodak® image station 4000 series
Kodak® Image Station 4000 Series, supplied by Kodak, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/kodak+4000+imager/kodak+image+station+4000+mm+pro/us11903675-171-93-97
Average 90 stars, based on 1 article reviews
kodak® image station 4000 series - by Bioz Stars, 2026-10
90/100 stars

Images

Related Articles

Electroporation:

Article Title: Single-cell Tsc1 knockout during corticogenesis generates tuber-like lesions and reduces seizure threshold in mice
Article Snippet: .. The region of electroporation was imaged using a Kodak 4000 imager. .. The next day, 100-μm-thick slices were prepared using a vibratome (Leica VTS 1000).

Article Title: Hypoxia-inducible factor 1a is a Tsc1-regulated survival factor in newborn neurons in tuberous sclerosis complex.
Article Snippet: .. Mice were prescreened for successful electroporation prior to sacrificing by viewing with an epifluorescence microscope or a Kodak 4000 imager. ..

Article Title: Hypoxia-inducible factor 1a is a Tsc1-regulated survival factor in newborn neurons in tuberous sclerosis complex.
Article Snippet: .. The region of electroporation was imaged using a Kodak 4000 imager. .. The next day, 100-mm-thick slices were prepared using a vibratome (Leica VTS 1000).

Article Title: Single-cell Tsc1 knockout during corticogenesis generates tuber-like lesions and reduces seizure threshold in mice
Article Snippet: The breeding lines were also crossed with the GFP fluorescent reporter line CAG-GFP (Jackson Laboratories; B6.Cg-Tg[CAG-Bgeo/GFP]21Lbe/J), in which, upon Cre expression, GFP is expressed. .. Mice were prescreened for successful electroporation prior to sacrificing by viewing with an epifluorescence microscope or a Kodak 4000 imager. ..

Mouse Assay:

Article Title: Hypoxia-inducible factor 1a is a Tsc1-regulated survival factor in newborn neurons in tuberous sclerosis complex.
Article Snippet: .. Mice were prescreened for successful electroporation prior to sacrificing by viewing with an epifluorescence microscope or a Kodak 4000 imager. ..

Article Title: Single-cell Tsc1 knockout during corticogenesis generates tuber-like lesions and reduces seizure threshold in mice
Article Snippet: The breeding lines were also crossed with the GFP fluorescent reporter line CAG-GFP (Jackson Laboratories; B6.Cg-Tg[CAG-Bgeo/GFP]21Lbe/J), in which, upon Cre expression, GFP is expressed. .. Mice were prescreened for successful electroporation prior to sacrificing by viewing with an epifluorescence microscope or a Kodak 4000 imager. ..

Microscopy:

Article Title: Hypoxia-inducible factor 1a is a Tsc1-regulated survival factor in newborn neurons in tuberous sclerosis complex.
Article Snippet: .. Mice were prescreened for successful electroporation prior to sacrificing by viewing with an epifluorescence microscope or a Kodak 4000 imager. ..

Article Title: Single-cell Tsc1 knockout during corticogenesis generates tuber-like lesions and reduces seizure threshold in mice
Article Snippet: The breeding lines were also crossed with the GFP fluorescent reporter line CAG-GFP (Jackson Laboratories; B6.Cg-Tg[CAG-Bgeo/GFP]21Lbe/J), in which, upon Cre expression, GFP is expressed. .. Mice were prescreened for successful electroporation prior to sacrificing by viewing with an epifluorescence microscope or a Kodak 4000 imager. ..



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In vivo targeting of Col IV to exposed basement membrane after induction of flow disturbance to recapitulate aspects of superficial erosion. (A) Immunohistochemical analysis of in vivo preparation of superficial erosion in mice. Microscopic observation reveals neo-intima formation (ni) and areas of denuded endothelium (red arrows) and exposed basement membrane (black dashed line). (Scale bar 100 µm). (B) A constrictive cuff was placed on previously injured and healed LCCA to perturb flow. Representative Evans Blue staining reveals increased endothelial permeability in areas of disturbed flow. <t>Fluorescence</t> <t>reflectance</t> images of RCCA (healthy carotid) and LCCA (‘eroded’ plaque) in mice injected with bare NPs and Col IV-NPs at 24h post-injection. (C) Quantitative assessment of fluorescence intensities of bare and Col IV-NPs reveals a 1.8-fold higher accumulation for Col IV-NPs to denuded endothelium compared to bare NPs (n = 3–4). (D and E) Immunofluorescent staining of CD31 (D) and Col IV (E) shows NP localization downstream of the flow perturbation. Col IV-NPs co-localize with areas of desquamation (white arrows in D) and rich in Col IV (E). (Scale bar = 25 µm). (F–H) Quantitative assessment of fluorescence intensities using <t>fluorescence</t> <t>reflectance</t> <t>imaging</t> analysis of bare or Col IV-NPs loaded with cy5.5 shows higher payload accumulation when delivered by Col IV-NPs at both 4 and 24 h after injection (n = 3–5): Tukey’s multiple comparisons test: **P < 0.01; ****P < 0.0001.
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In vivo targeting of Col IV to exposed basement membrane after induction of flow disturbance to recapitulate aspects of superficial erosion. (A) Immunohistochemical analysis of in vivo preparation of superficial erosion in mice. Microscopic observation reveals neo-intima formation (ni) and areas of denuded endothelium (red arrows) and exposed basement membrane (black dashed line). (Scale bar 100 µm). (B) A constrictive cuff was placed on previously injured and healed LCCA to perturb flow. Representative Evans Blue staining reveals increased endothelial permeability in areas of disturbed flow. <t>Fluorescence</t> <t>reflectance</t> images of RCCA (healthy carotid) and LCCA (‘eroded’ plaque) in mice injected with bare NPs and Col IV-NPs at 24h post-injection. (C) Quantitative assessment of fluorescence intensities of bare and Col IV-NPs reveals a 1.8-fold higher accumulation for Col IV-NPs to denuded endothelium compared to bare NPs (n = 3–4). (D and E) Immunofluorescent staining of CD31 (D) and Col IV (E) shows NP localization downstream of the flow perturbation. Col IV-NPs co-localize with areas of desquamation (white arrows in D) and rich in Col IV (E). (Scale bar = 25 µm). (F–H) Quantitative assessment of fluorescence intensities using <t>fluorescence</t> <t>reflectance</t> <t>imaging</t> analysis of bare or Col IV-NPs loaded with cy5.5 shows higher payload accumulation when delivered by Col IV-NPs at both 4 and 24 h after injection (n = 3–5): Tukey’s multiple comparisons test: **P < 0.01; ****P < 0.0001.
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Image Search Results


In vivo targeting of Col IV to exposed basement membrane after induction of flow disturbance to recapitulate aspects of superficial erosion. (A) Immunohistochemical analysis of in vivo preparation of superficial erosion in mice. Microscopic observation reveals neo-intima formation (ni) and areas of denuded endothelium (red arrows) and exposed basement membrane (black dashed line). (Scale bar 100 µm). (B) A constrictive cuff was placed on previously injured and healed LCCA to perturb flow. Representative Evans Blue staining reveals increased endothelial permeability in areas of disturbed flow. Fluorescence reflectance images of RCCA (healthy carotid) and LCCA (‘eroded’ plaque) in mice injected with bare NPs and Col IV-NPs at 24h post-injection. (C) Quantitative assessment of fluorescence intensities of bare and Col IV-NPs reveals a 1.8-fold higher accumulation for Col IV-NPs to denuded endothelium compared to bare NPs (n = 3–4). (D and E) Immunofluorescent staining of CD31 (D) and Col IV (E) shows NP localization downstream of the flow perturbation. Col IV-NPs co-localize with areas of desquamation (white arrows in D) and rich in Col IV (E). (Scale bar = 25 µm). (F–H) Quantitative assessment of fluorescence intensities using fluorescence reflectance imaging analysis of bare or Col IV-NPs loaded with cy5.5 shows higher payload accumulation when delivered by Col IV-NPs at both 4 and 24 h after injection (n = 3–5): Tukey’s multiple comparisons test: **P < 0.01; ****P < 0.0001.

Journal: Cardiovascular Research

Article Title: Targeted delivery of protein arginine deiminase-4 inhibitors to limit arterial intimal NETosis and preserve endothelial integrity

doi: 10.1093/cvr/cvab074

Figure Lengend Snippet: In vivo targeting of Col IV to exposed basement membrane after induction of flow disturbance to recapitulate aspects of superficial erosion. (A) Immunohistochemical analysis of in vivo preparation of superficial erosion in mice. Microscopic observation reveals neo-intima formation (ni) and areas of denuded endothelium (red arrows) and exposed basement membrane (black dashed line). (Scale bar 100 µm). (B) A constrictive cuff was placed on previously injured and healed LCCA to perturb flow. Representative Evans Blue staining reveals increased endothelial permeability in areas of disturbed flow. Fluorescence reflectance images of RCCA (healthy carotid) and LCCA (‘eroded’ plaque) in mice injected with bare NPs and Col IV-NPs at 24h post-injection. (C) Quantitative assessment of fluorescence intensities of bare and Col IV-NPs reveals a 1.8-fold higher accumulation for Col IV-NPs to denuded endothelium compared to bare NPs (n = 3–4). (D and E) Immunofluorescent staining of CD31 (D) and Col IV (E) shows NP localization downstream of the flow perturbation. Col IV-NPs co-localize with areas of desquamation (white arrows in D) and rich in Col IV (E). (Scale bar = 25 µm). (F–H) Quantitative assessment of fluorescence intensities using fluorescence reflectance imaging analysis of bare or Col IV-NPs loaded with cy5.5 shows higher payload accumulation when delivered by Col IV-NPs at both 4 and 24 h after injection (n = 3–5): Tukey’s multiple comparisons test: **P < 0.01; ****P < 0.0001.

Article Snippet: After perfusion, both carotids were collected and visualized using the fluorescence reflectance imaging (FRI) system (Kodak Image-Station 4000, Carestream Health, Rochester, NY, USA).

Techniques: In Vivo, Membrane, Immunohistochemical staining, Staining, Permeability, Fluorescence, Injection, Imaging